• Are they symptomatic?
  • Vitals, EKG, prior EKG
  • Always see the patient!
    • Unstable = chest pain, respiratory distress, new confusion, new hypotension
      • → ACT, ACLS algorithm; set up for EKG, defibrillation/cardioversion

Pulseless

  • CPR - minimize interruptions
  • Shockable rhythms -  VT, Vfib
    • Vtach
    • Vfib
  • Meds
    • Epi 1 mg q3
    • Amio 300 mg for refractory VT/Vfib
  • Reversible causes
    • H = hypovolemia, hypothermia, hypoglycemia, [H+] acidosis, hypo/hyperK
    • T = tension pneumo, tamponade, toxins, thrombosis (PE/MI)

Sinus

  • Tachy/brady
    • Treat underlying causes - pain, hypoxemia, sepsis, HF, PE, thyroid, meds
    • BB tox = IV glucagon
    • CCB tox = IV calcium
  • Sinus arrhythmia = normal; nothing to do
  • Sinus pause = may be blocked PAC; look for P wave buried in the preceding T wave (T wave will have diff morphology). AV node is refractory so following complex is not conducted. Tx with BB

Atrial arrhythmia

  • Atrial fibrillation/flutter
    • Rate = 150 may be A flutter 2:1
  • Wandering atrial pacemaker = benign, nothing to do
  • Multifocal atrial tachycardia
    • Often in setting of lung disease; treat underlying issues (no need to anticoagulate)
    • Verapamil or metoprolol

AV junction arrhythmia

  • ???

Supraventricular tachycardia

  • AVNRT - retrograde P wave?
    • Vagal maneuvers
    • Adenosine - start with 6 mg, then 12 mg if needed - warn pt feels like kick in chest/heart stopping for few seconds
      • Push and flush rapidly (very short half-life)
    • Continuous rhythm strip while trying therapy
    • Avoid AV nodal blocking meds (BB, CCB, adenosine, digoxin, amiodarone) if afib with pre-excitation (WPW) (FBI = fast, broad, irregular)
      • Give procainamide instead
    • Unstable = cardiovert
  • AVRT
  • Junctional tachycardia

AV block

  • If bradycardic + symptomatic, likely Type II 2nd degree or complete AVB
    • Pacemaker (transcutaneous -> transvenous); sedate before if possible
   

1st degree

  • Prolonged PR > 200 ms
 
2nd degree
  • Mobitz type I (Wenckebach) = progressively longer PR before QRS drop
  • Mobitz type II = sudden dropped QRS; may progress to 3rd degree AVB (due to disease below AV node)
Wenckebach
Mobitz II

3rd degree

  • Complete block; dissociation of P and QRS
  • Atrial and ventricular rates are independent
3rd degree heart block

 

Ventricular arrhythmias

  • Accelerated idioventricular rhythm - look for acute MI. Not VT bc rate < 100

Wide complex ventricular tachycardia

  • HR > 100, QRS > 120 ms
  • Regular ~ VT, chaotic ~ Vfib
  • Check for hemodynamic stability - hypotension, chest pain, mental status
    • Unstable = cardiovert; have pads on if uncertain
    • Stable = amio bolus; elective synchronized cardioversion
  • If pt has known heart disease, treat as VT
  • Check K, Mg (want K > 4, Mg > 2)

Ventricular tachycardia

  • Extreme tachycardia + loss of coordinated atrial kick → decreased cardiac output
  • Can degenerate into vfib
  • Commonly seen in setting of previous heart disease/poor ventricular function (MI, CHF, etc.)
  • Regular rhythm with AV dissociation, very broad QRS > ~ 160 ms
  • Monomorphic VT - QRS complexes are uniform
  • May see 'capture'/'fusion' beats that occur when atrial and ventricular contractions line up/coincide - have narrow complex or hybrid morphology

Vtach

Evaluation/tx

  • Sustained (> 30s or requiring intervention)
    • Cardioversion
  • Non-sustained (NSVT) (≥ 3 complexes terminating in < 30s)
    • Not uncommon in cardiomyopathy - isolated NSVT not an emergency
    • Get echo if no prior h/o reduced EF
    • If frequent, consider ongoing myocardial ischemia
    • Consider giving BB if OK
    • Keep K > 4, Mg > 2

Polymorphic VT/Torsades de pointes

  • Form of VT with multiple ventricular foci → multiple QRS morphologies
  • Torsades = polymorphic VT with QT prolongation → complexes 'twist' around the isoelectric line with increasing/decreasing amplitudes 
  • May degenerate into Vfib

Torsades 

Evaluation/tx

  • If stable, give IV Mg sulfate 2 mg, correct long-QT
  • If unstable, defibrillate

 

 

 

Ventricular fibrillation

  • Ventricles do not contract in synchronized manner → loss of cardiac output
  • Chaotic, no identifiable conformations, varying amplitude that decreases with prolonged vfib → asystole
  • Rate 150-500 bpm
  • Look at the rhythm strip, not the 12-lead to diagnose

Vfib
Evaluation/tx

  • CPR, defibrillate

 

  • Narrow complex
author: last edited: June 14, 2018, 1:17 p.m. | pk: 98 | unpublished